Thursday, 22 November 2012

Atherosclerosis - Part One


Atherosclerosis is sometimes referred to as “hardening of the arteries.” It is the end result of a process of fatty infiltration and later calcification of the walls of the arteries (which simply means that fatty deposits called plaques build up in the walls of the arteries and become hard). This process is well-known for its silent role in heart attacks and strokes. There are usually very few signs of this process occurring, and it’s only gradually suspected when an individual gets a high blood cholesterol reading from a blood test; even that can only be suspected. The process of atherosclerosis can start in childhood so it is always a good time to prevent it.
The major risk factors are high cholesterol levels and high blood pressure. An improper diet is a major cause of atherosclerosis. Saturated fats (found mostly in animal fats), deep-fried food, too much meat and too little fibre, low fruit and vegetable intake will have serious consequences. Coffee and caffeine drinks and alcohol are high on the danger list. A diet high in sweets, fast foods and refined foods will lead to atherosclerosis. Homogenised milk is responsible as well, since fat particles in the milk are more likely to cause plaques to form in the arteries. Tobacco is another substance that hinders circulation and is hard on the arteries. Lack of exercise increases the risk of developing atherosclerosis. A high-stress lifestyle leads to high blood pressure and increases the risk of cardiovascular accidents like heart attacks and strokes. It must be said that cardiovascular disease is the single most important cause of death in the Western world. In 1900, only 1 in 7 people died of cardiovascular disease, and only 1 in 30 people died of cancer. Today 45% of deaths are due to cardiovascular disease while cancer accounts for 25% of deaths. What has changed so much? Diet and lifestyle, I believe, are among the major factors involved. For effective prevention of both of these diseases, our diet must be high in fruit and vegetables, fish and dietary fibre. Simply reducing the intake of fats and cholesterol is inadequate. The amount of dietary cholesterol does not necessarily correspond with the level of cholesterol in the blood, and it is not the amount of fat we eat but rather the types of fat we eat and the way we metabolise them that are important.
Saturated fatty acids found in meat and animal products are two main culprits in the build up of plaques and the clogging of arteries. Animal proteins raise cholesterol levels, while vegetable proteins lower them. Equally harmful are processed, trans-fatty, hydrogenated fats like margarine and shortening and the heat-damaged fats of deep-fried foods. They interfere with the two essential fatty acids, Omega 3 and Omega 6 (“essential” means they have to be added to our diet as we can’t make these oils ourselves).
These essential oils, Omega 3 and Omega 6, are found in unrefined, cold-pressed nut and seed oils and are very important to the prevention of fatty degeneration (which leads to the build up of fatty deposits in our arteries). They lower the level of blood cholesterol, inhibit platelet aggregation (which causes blood clots to form leading to heart attacks and strokes) and are therefore essential weapons in the fight against atherosclerosis. The best source of both essential oils is unrefined, cold-pressed, flax- seed oil.
The fatty acids in flaxseed oil are converted in our bodies into EPA, which is also found in certain fish oils. EPA is present in cold-water fish like salmon, mackerel and herring. These oils are powerful weapons against platelet aggregation. Taking as little as 20ml (two tablespoons) of flaxseed oil a day in only two weeks doubles blood levels of EPA, resulting in a marked decrease in platelet aggregation and therefore reduces the formation of atherosclerosis.
Next week I’ll again look at atherosclerosis and methods of ensuring that this number one killer in the Western world can be avoided.

David Foley
MNIMH, MRCHM
Medical Herbalist

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